cjc 1295 dac vs no dac, is the half life really the whole story?
Trying to state this cleanly because the naming is a mess.
CJC-1295 with DAC has a drug affinity complex that binds albumin. Half life goes from minutes to days.
CJC-1295 without DAC is basically Mod GRF(1-29), a short acting GHRH analog. Half life measured in minutes.
So with DAC you get a sustained elevation, no DAC you get something closer to a pulse. GH release is naturally pulsatile, and the argument for the short version is that it preserves that pattern instead of flattening it.
What I can't resolve from the literature: does the pulsatile pattern actually matter for anything people care about? It matters a lot in the underlying GH physiology. Whether it matters for outcomes is a different question and I don't think it's settled.
The half life numbers are solid. The "pulsatility matters so short acting is better" step is inference, not a demonstrated outcome difference. Am I wrong?